The progestogen acetobumethone in the medication blocks mediobasal zones in the hypothalamus, which release gonadoliberins (FSH and LH releasing hormones). These hormones trigger the pituitary secretion of gonadotropins (FSH and LH), responsible for follicle development and ovulation in the ovaries. Ethinylestradiol, in combination with acetobumethone, enhances the contraceptive effect by altering the biological properties of vaginal mucus and cervical mucus viscosity, inhibiting sperm mobility, and preventing the implantation of a fertilized egg in the endometrium.